Should a uric acid level of 550 be treated?

Update Date: 2024-05-29 Source: Network

Generally, uric acid levels of 550 require treatment.

This is because a uric acid level of 550 falls under the category of hyperuricemia. According to relevant treatment guidelines for hyperuricemia, when blood uric acid levels exceed 540umol/l, it is necessary to use uric acid-lowering drugs for treatment. Common uric acid-lowering drugs include benzbromarone, allopurinol, and febuxostat, among others. It is recommended that patients follow their doctor's advice and undergo treatment as prescribed. If uric acid levels of 550 are not treated, it can lead to gout attacks, affect metabolism such as blood sugar and blood lipids, and contribute to hypertension and hyperlipidemia.

Four symptoms of hyperuricemia:

1. Gout: If hyperuricemia remains uncontrolled, it can lead to the occurrence of gout. When uric acid salts accumulate continuously in the body and cannot be normally excreted, it can cause gout symptoms. Therefore, the higher the blood uric acid level, the greater the risk of gout.

2. Hypertension: High blood uric acid is also one of the predisposing factors for hypertension. Therefore, if blood uric acid levels continue to rise, it will increase the risk of developing hypertension. Clinically, patients with primary hypertension often have comorbid hyperuricemia, which also indicates that there is a causal relationship between hyperuricemia and primary hypertension.

3. Diabetes: The occurrence of hyperuricemia can easily lead to the destruction of patients' insulin function, which can cause diabetes. Investigations have found that long-term hyperuricemia is closely related to the onset of diabetes.

4. Coronary heart disease: Hyperuricemia can also often lead to the occurrence of coronary heart disease. High uric acid is one of the risk factors for high mortality rates in coronary heart disease, so hyperuricemia can pose a certain threat to patients' lives.

Hazards of hyperuricemia:

1. Gouty arthritis: Hyperuricemia is the fundamental cause of gout. The higher the blood uric acid level, the more frequent gout attacks and the earlier onset age.

2. Gouty nephropathy and chronic renal failure: High uric acid can damage the kidneys, and when kidney function is impaired, uric acid excretion decreases, which can further exacerbate hyperuricemia. For every 60μmol/L increase in blood uric acid, the risk of acute renal failure increases by 74%.

3. Uric acid kidney stones: According to statistics, 5.1% of patients with kidney stones in China have uric acid kidney stones. Like kidney stones caused by other reasons, uric acid nephrolithiasis often manifests as low back pain and hematuria. When the ureter is blocked, symptoms such as fever, oliguria, anuria, hydronephrosis, and elevated serum creatinine may also occur.